This would be consistent with the fact that in processes, such aswound healing, inflammation, and cancer, PPARδ levels seem to increase concomitantly withupregulation of cyclooxygenase-2 and other enzymes for the production of lipidmetabolites capable of stabilizing and activating PPARδ [32, 37, 55, 58, 59]. The gene discussed is PPARD; the disease is cancer.