Although expression of the Dbp molecules restored binding of the spirochetes to purified decorin, dermatan sulphate, and human epithelial cells [18], experiments to test the infectivity of B314 expressing DbpA and DbpB in the murine model of Lyme disease were implausible due to the loss of plasmids that are required for mammalian infection [39]. The gene discussed is DBP; the disease is infection.