Thus, it is proposed that subjects at high risk for CHD have increased hs-CRP, MPO, Lp-PLA2, LP, IL-6, TNF-α, LTB4 and low levels of various PUFAs, LXs, resolvins, protectins, and nitrolipids, and the balance between these pro-inflammatory and anti-inflammatory molecules may also aid in the prediction and prognosis of CHD. This evidence concerns the gene TNF and coronary artery disorder.