In our study, the mutant genotype (CC) frequency of T-786C polymorphism was found in low percentage (5%) to that of wild type (TT) (75%) and heterozygous (CT) (20%), which indicates that no association persists between T-786C variation and CHD whereas, the frequency distributions of eNOS T-786C genotypes were similar in CHD and control groups in the present study. This evidence concerns the gene NOS3 and coronary artery disorder.