In the current study, apart from correlating these putative p53-miRs with the upstream regulators and downstream targets of p53, we prioritize our predictions using (a) literature reported differentially expressed miRNAs in cancer; (b) miRNAs reported as induced or repressed following p53 activation [11]; and (c) miRNA targets that are functionally enriched with biological processes like apoptosis, cell cycle. Here, TP53 is linked to cancer.