Thus, a recent studydemonstrated significantly elevated MCP-1 concentrations in preeclampticpatients [37], whereas other researchers found no significant differences in plasma MCP-1 concentrations between preeclamptic and normal pregnant women [38].In the former study [37], it has been hypothesized that, in preeclampsia, the damaged endothelial cells and/or infiltrated macrophages may produce MPC-1, which probably promotes extension of the lesions. The gene discussed is CCL2; the disease is preeclampsia.