The observed shifts in glycosylation were not due to pre-dysplastic alterations of the gastric mucosa such as intestinal metaplasia, because the spatial distribution of expression patterns for the two common gastric mucins MUC5AC and MUC6 (Figure S3) were unchanged during the 10-month period of experimental infection, with no aberrant MUC2 or atypical sulfo-mucins being detected (data not shown). Here, MUC6 is linked to infection.