Many genes in the biosynthesis (papA5, ppsC, pks1, pks15, fadD22/29) and export (drrB-C, lppX) [51], [52] of phtiocerol dimycocerosate (PDIM), a surface molecule that is important in pathogenesis, were also more weakly expressed in the M. tuberculosis response to DC compared to Mφ infection. This evidence concerns the gene ARAFP2 and infection.