Consistent with this conclusion, we have recently published in collaboration with the Kenney laboratory data that indicate the following: (i) ZEB1 levels range from very high to moderate to absent among a variety of epithelial and B-cell types that are physiologically relevant to EBV; (ii) The correlation between ZEB1 abundance and whether infection by EBV is latent or lytic is fairly good, but far from perfect; and (iii) The abundance of activated c-Jun also contributes to how a cell responds to EBV infection [38]. The gene discussed is JUN; the disease is infection.