Unlike evidence from experimental studies in which the affinity to aryl hydrocarbon receptor (AhR) was important to induce atherosclerosis (Hennig et al. 2002; Stegeman et al. 1995; Toborek et al. 1995), the strengths of association of each POP belonging to the category of PCDDs or PCDFs did not appear to be correlated with the toxic equivalent factors (TEFs) of each POP. Here, AHR is linked to atherosclerosis.