Molecular profiling of transformed and metastatic murine SCCs showed that Gro1 (IL8 homolog), antiapoptotic gene cIAP-1 (cIap-1/Birc2), and Yap65 (YAP 1) were upregulated and clustered together in association with activation of NF-κB and tumor growth, metastatic progression, and angiogenesis [37,38]. Here, NFKB1 is linked to neoplasm.