ERBB2 and cancer: Our findings revealed that exogenous supplementation of cultured cancer cells with physiological concentrations of the ω-9 MUFA OA drastically suppressed the expression and activity of HER2 (erbB-2) [9,11,19-21], one of the most commonly analyzed proto-oncogenes in human cancer studies as it plays a pivotal role in oncogenic transformation, tumorigenesis and metastasis [22-25].