In line with results in chronic HIV-1 infection [16], [17], [26], no statistical difference was observed between either breadth or magnitude of the HIV-1-specific T cell response as measured by IFN-γ-production in HIV-1 Controllers and Progressors (p = 0.5 and p = 0.21, respectively), as previously reported for a small subset of these study subjects [14], [15]. This evidence concerns the gene IFNG and HIV-1 infection.