Our results show that in half of the clinical specimens, liver metastases had a much higher expression of CEACAM6 than the primary colorectal tumors, suggesting that in such patients, blocking adhesion and invasion that results from CEACAM6 expression might have influenced the ability of tumor cells to metastasize, as we have in fact shown experimentally [4]. The gene discussed is CEACAM6; the disease is colorectal neoplasm.