Rotavirus infection has already been associated with pancreatic islet autoimmunity through a mechanism of molecular mimicry [34–36] between viral peptide sequences, in particular within the VP-7 protein and T cell epitopes in the islet cell autoantigen glutamate decarboxylase (GAD) and tyrosine phosphatase IA-2 [37,38]. The gene discussed is GLUL; the disease is Rotavirus infection.