The two molecular forms of autosomal dominant Dunnigan-type familial partial lipodystrophy (FPLD) result from mutations either in LMNA encoding nuclear lamin A/C (FPLD2; MIM 151660) or in PPARG encoding peroxisome proliferator-activated receptor-γ (FPLD3; MIM 604367) [1-3]. Here, PPARG is linked to familial partial lipodystrophy.