Binding and Neutralising Anti-IFN Autoantibodies in APS1 Patients with AIRE Mutations, in their Unaffected Heterozygous Relatives, and in Patients with MG/Thymoma (as a “Positive” Control Group), with APS2, or with Sporadic Diseases from the APS1 Triad. This evidence concerns the gene IFNA1 and autoimmune polyendocrine syndrome type 1.