Airway inflammation and alterations in cellular turnover are histopathologic features of asthma [4] and recently, research has disclosed the involvement of PPARs such as PPAR-γ and PPAR-α in many facets of the disease such as decreasing antigen-induced airway hyperresponsiveness, lung inflammation, eosinophilia, cytokine production and serum levels of antigen-specific IgE [29]. This evidence concerns the gene PPARG and Increased total eosinophil count.