BCR and neoplasm: Different tumour phenotypes can result from specific versions of related fusion proteins, for instance, the Philadelphia t(9;22) translocation (which yields the BCR-ABL [breakpoint cluster region gene–Abelson leukaemia oncogene] fusion) occurs in both myeloid- and lymphoid-lineage tumours, dictated by the position of the translocation junction within the BCR gene [5].