DCs that had internalized dying cells were tested for their ability to cross-present antigen; in contrast to their ability to present antigen via the “classical” MHC I pathway, the TAP−/− DCs were able to cross-present influenza antigen derived from MHC-mismatched apoptotic cells as efficiently as WT DCs (Figure 1D and 1E). The gene discussed is HLA-C; the disease is influenza.