FAS and pulmonary fibrosis: Alterations in the susceptibility of these cells to Fas-induced cell death contribute to the pathogenesis of lung fibrosis, [6,7] and myofibroblasts are susceptible to the suppression of apoptosis by transforming growth factor-β1 (TGF-β1) [6] and resistant to interleukin (IL)-6-induced apoptosis [8].