CGA’s robust, highly homogeneous consensus VF profile (F16 papA allele, papG allele II, iutA, kpsM II, traT, and ompT) suggests considerable extraintestinal virulence potential, an inference supported by experimental data indicating that CGA is able to compete successfully with classic group B2-derived pathogens in a mouse UTI model (J.R. Johnson, unpub. Here, PAPOLG is linked to bacterial urinary tract infection.