The initial microarray data suggested that S100A2 was also over-represented in eight out of nine of these particular tumours, but there were a further 10 lesions in which S1000A2 was strongly expressed in the apparent absence of TP73L transcript over-representation and one lesion in which TP73L was marginally increased with no apparently corresponding upregulation of S100A2. Here, TP63 is linked to neoplasm.