CEACAM6 overexpression is associated with greater resistance to anoikis and increased metastatic potential, whereas post-transcriptional inhibition of CEACAM6 expression impairs the ability of pancreatic adenocarcinoma cells to resist anoikis and to form experimental liver metastases in vivo (Duxbury et al, 2004b). This evidence concerns the gene CEACAM6 and pancreatic adenocarcinoma.