This indicates possible mutation of p53 as an early event, which is typical of the carcinogenic pathway for non-endometrioid carcinomas.19-21 Conversely, estrogen and progesterone receptors are more frequently positive in endometrioid carcinoma, particularly if well-differentiated, as reported by others.9,18,21-23 However, in our results, only progesterone receptor expression was statistically significant, which could possibly be explained by the antibodies coming from different sources or having different sensitivity and specificity, as reported by others.23 The gene discussed is PGR; the disease is endometrioid adenocarcinoma.