To overcome their observed binding of normal AGP (which by definition will have a different glycosylation pattern from CML-derived AGP and be of limited clinical relevance) to imatinib, it was suggested using clindamycin or erythromycin concomitantly, since the antibiotics purportedly compete with imatinib for AGP-binding sites. Here, ATP5MK is linked to chronic myelogenous leukemia, BCR-ABL1 positive.