LGALS3 and Miyoshi myopathy: In addition, MM cells were found to express the high-affinity laminin binding protein 67LR, which appeared to be upregulated by contact with the endothelium, indicating that during passage through the basement membrane, 67LR is contemporary upregulated allowing MM cells to be sensible to the chemoattractive properties of laminin-1, and so facilitating transendothelial migration.